Wednesday, March 11, 2009

TQT Time


Submit your TQTs based on the chapters 19 and 21 notes as comments to this post.

Mr. Baker


Ca said...

1. How come excessive cell division can result from mutations in the ras proto-oncogenes?
2. How does a mutation in tumor-suppressor genes causes cancer?
3. Why does a mutation knocking out the p53 gene can lead to excessive cell growth and cancer? What exactly are the causes of this?

TahiraMadness said...


1) So cancer is basically a cell that's grown too big and then divided rapidly? How does it harm the body? Aren't there 2 types of tumors?

2) In morphogenesis for flies they go from being that wormy thing to a fatter wormy thing then a fly, do humans go through any sort of morphogenesis? Or does sitting in a womb for a million years not count?

3) If the bicoid gene determines where the body segments go has it ever shifted and then because of that got creatures with arms sticking out of the wrong places? Or does that never happen and am I being crazy?



Anonymous said...

Is gene amplification random?

are all cells in a multicellular organism the same until signals make them turn genes on and off making them specialized?

are there species in which the vast amounts of repeated DNA are fundamental?

Ana said...

1. I dont understand how the segmentation pattern is set up and how the mutations cause the gaps in the segmentation.

2. The book says that high levels of inducer produces division and differentiation in the vulva and activation of a gene. Does it only work when the inducer is at a high level what if it was at a low level?

3.How does the organ idenity genes work in plants? IF there are 3 classes of organ identity genes does each work in the same way but in different places.

Bulgan said...

1. What does it mean when the cell is a secretory cell?

2. What is the hypothesis saying about the BRCA1 and BRCA2, because it says "one or both proteins are involved in DNA repair.

3. Can viruses exist in human's body but do not cause any cancer or harm to the host?

Inwoo said...

1. The cloning of a sheep was really amazing to me; do we have the ability to clone humans too?
2. If a species, such as the drosophila, have such a short life cycle, isn't there a higher chance for a higher rate of evolution for them than humans?
3. Apoptosis, is it a "suicide" or just itself dying out?

Shauna said...

umm are we not having a TQT this week or am I totally lost?

ZS said...

1. In nucleus, why moledules of a cytoplasmic determinant skew to one of the two-celled embryo, after mitotic cell division? what does that do? is there going to be an empty cell that has absence of molecules of a cytoplasmic determinant?
2. how embryonic lethals works?
3. How segmentation pattern works?